Kevin M. Hannon
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Contact Information Email: hannonk |at| purdue.edu Office: LYNN G138 Map Phone: (765) 494-5949 Fax: (765) 494-0781 |
Associate Professor of Basic Medical Sciences |
Education
B.S. Purdue University 1985
M.S. Iowa State University 1987
Ph.D. Iowa State University 1990
Research Interests
Developmental biology, growth factor biology, and molecular biology Our lab studies the molecular events that regulate the decision of a pluripotential stem cell to commit to a specific myogenic {skeletal muscle}, chondrogenic {cartilage}, or osteogenic {bone} lineage in vivo and in vitro
Teaching Interests
Veterinary Anatomy
Grants
Musculor Dystrophy Association
IAMs
Veterinary Anatomy
United States Department of Agriculture
Publications
Flanagan-Steet*, H., K. Hannon*, M. McAvoy, R. Hullinger, B. B. Olwin. (2000). Loss of FGF Receptor 1 Signaling Reduces Skeletal Muscle Mass and Disrupts Myofiber Organization in the Developing Limb. Developmental Biology, Vol. 218, No. 1, pp. 21-37 (*indicates equal contributors, listed alphabetically).
Clase, K. P., Mitchell, P. Ward, C. Dorman, S. Johnson, and K. Hannon. (2000). FGF5 Stimulates Connective Tissue Development and Inhibits Skeletal Muscle Development in the Limb: Developmental Dynamics: 219:368-380.
Mitchell, P., S.E. Johnson and K. Hannon. (2002). IGF-1 Stimulates Primary Myofiber Development During Limb Development. Developmental Dynamics: 223:12-23.
Ward, P, K. Clase and K. Hannon. (2003). FGF10 Stimulates myogenesis in vitro but not in vivo. Journal of Animal and Veterinary Advances: 2(4) 196-201.
Alzghoul, M.B., D. Gerrard, B.A. Watkins and K. Hannon. (2004). Ectopic Expression of IGF-I and Shh by Skeletal Muscle Inhibits Disuse-Mediated Skeletal Muscle Atrophy and Bone Osteopenia In Vivo.2004 FASEB J. Jan; 18(1): 221-3. Epub 2003 Nov 03.